PRESS RELEASE This contents of this page was originally posted in some closed forums since July 30, 2007 And is now made public on my website on October 9, 2008. Lehigh Valley, Pennsylvania U.S.A. By Paul J. Tubiana
An open request for new research:
Medical Students, Medical Doctors, Nurses, Health practitioners in any capacity, Medical and Scientific Researchers, Diabetes Educators, Nutritionists, Chemists, Biologists, Microbiologists, Cellular Microbiologists, Biochemists, and ambitious scientists in any capacity who feel capable, public officials, leaders in the public and private sector
To study, research, find, advocate for and publish information that confirms what is presented below in order
to Cure Diabetes:
FOR IMMEDIATE RELEASE
A Request for More Specific Research into Diabetes.
Many basic questions about diabetes have remained unasked and unanswered over the past thirty years. Research communities involved in direct research to find a cure for diabetes need to work closer with the community of diabetics and try to give answers to their claims and complaints. I discovered after twenty-one years of believing, as I was told, to have a "Type 1" diabetes condition, that I had no such condition and that I had in fact a Type 2 / Insulin Resistance condition. This discovery was only possible thanks to my first C-Peptide test which had always been available during my diabetic lifetime, but was only performed twenty-one years after I was diagnosed. This raised some serious questions in my mind about where diabetes research has gone and continues to go when such a basic question has been so prominently ignored. I feel that there are many "Type 1's" like myself that are really Type 2's? If there are others who feel they were misdiagnosed, I invite them to sign my petition that we are gathering for more exact testing at http://www.petitiononline.com/CPT12345/petition.html
I feel that the diabetic community would benefit greatly from specific research into these areas which would foster prevention and finding a practical cure:
1. Individualized Stimulated C-Peptide testing along with a blood glucose test at the same time, to be done in all cases involving presumed Type 1 Diabetes / Juvenile Diabetes / Insulin Dependent Diabetes. The C-Peptide test should be done under "non-fasting" conditions and if the blood glucose is in the normal range, the results should be considered non-conclusive.
2. In non-diabetic cases only with a known family history or risk factors such as obesity and/or sedentary lifestyle, more Fasting C-Peptide testing along with a blood glucose test to be used as a screen for insulin resistance. If the blood glucose is above normal the results should be considered non-conclusive. Increased C-Peptide levels with normal blood glucose should be an indicator of a development of insulin resistance.
3. All previous studies that link X to Type 1 Diabetes should be reevaluated to see if they also connect to Type 2 diabetes. Based on my experience, for example: Study on Milk and Type 1 Diabetes could yield information for a direct connection for Milk and Type 2 Diabetes.
4. It is my opinion that until 2004 Type 1 Diabetes has been just a label which was not, in most cases based on fact. It is not clear yet whether more or less C-Peptide testing is actually being conducted to clarify the diagnosis. Most funding for diabetes research both public and private has traditionally gone for a condition known as Type 1 diabetes while most diabetics have a Type 2 or Insulin Resistance medical condition. In light of my experience, perhaps more people labeled as having Type 1 may actually have Type 2. I think the proper diagnosis of all diabetics is long overdue and the individual results should determine where our precious resources go to fund relevant research.
5. I would like to know why when I took Penicillin based antibiotics, I was able to completely get off insulin for a day and have normal blood glucose levels. This would not have been possible through diet alone. It is well known that the mechanism of action for Penicillin is to act on the cell wall of bacteria while they multiply, causing the bacteria to explode. What effect does Penicillin have on human physiology in general and on the physiology of human cells?
6. More research into the mechanism of Insulin Resistance, this should involve the fields of Molecular Biology, Biochemistry, Cell Science, Microbiology, etc. Specifically, the role of PROTEINS causing a malfunction of the insulin receptor should be explored. For this purpose the role of insulin as a protein causing a log jam in the insulin receptor (Most insulins are chemically not identical to natural human insulin). It is my experience and theory that insulin acts as a key which part of it breaks off in the lock, causing it to act as plug. The only way to clear this plug out of the way of the lock is to introduce more insulin or push out the old piece of the key by pushing in a new key. The role of Penicillin and its possible effects on the cell mechanism causing insulin resistance should be explored. The role of bacterial enzymes and toxins on human physiology and the physiology of human cells should be explored. The following explanations that I received from Dr. Johann Georg Schnitzer by e-mail should also be further researched.For information see his website:http://www.doc-schnitzer.comor http://www.dr-schnitzer.de “Diabetes Type II is caused by widespread wrong feeding habits, which were and are introduced, trained and maintained by groups which either live upon the wrong foodstuff side, or upon the resulting diseases side. In the table of Homotoxicology according to Dr. Hans Heinrich Reckeweg, which you find in my book about diabetes, you could localize it as a "deposition phase" (it's mainly a deposition of mucopolysaccharides in the basal membrane of the cardiovascular system, in the interstitium and in the membranes of all cells - provided by an oversupply with animal protein and refined carbohydrates from wrong feeding habits).” “Milk causes mucus production. Mucus is an ideal soil for growth of bacteria. Glucose: E.g. Poliomyelitis only can infect a body after a sugar spike, which is followed by high insulin production and therefore a hypoglycemia (which opens the door widely for the virus). But milk also increases the oversupply with protein, which causes cardiovascular and other diseases.” “The effect of protein oversupply isn't a direct one like eating sugar; it's an indirect one. By a partial storage of protein in the cellular membranes, in the interstitium, and in the basal membrane of the capillaries, the transport way for insulin is increased up to 15 times longer, what makes the transport time for insulin from the B-cells to the insulin receptors on the surface of the cells 15 x 15 = 225 times longer. In addition, the ability of the insulin receptors becomes considerably impaired.”
7. The role of nutrition and the knowledge concerning food science have all been completely ignored by medical research as contributors to causing diabetes. The role of genetics has been cleverly exploited to keep diabetics from either seeking or asking important and relevant questions. Clearly, the current explosion of diabetes around the globe gives proof that this is more than just a genetic condition. In fact it was said not too long ago that diabetes was a disease that affected mostly industrialized countries. If that was the case, I cannot understand the constant fervor over genetics while ignoring basic questions. My opinion on the matter is this: All people have a specific genetic capacity to produce insulin, of course those who produce the least will be first in line to suffer the effects of insulin resistance or to develop diabetes. But in my case it should be clear to everyone that there is more insulin in my body than any normal pancreas could produce, therefore in my opinion all people are at risk for insulin resistance even if it never manifests itself as diabetes. Many medical conditions are attributable to elevated or high C-Peptide levels which is an indicator of insulin resistance. Roses do well in full sun but not in shade. If the genetic argument of diabetes were applied to Roses, then it would be said that Roses have a genetic predisposition to die in the shade. No one would question that it is because of habitat and not genetics. For diabetes, I think we need to look at which habitat in the form of proper diet, nutrition, and lifestyle makes for the best use of the genetics we were given, just as we never question that all plants have their native habitat were they thrive and do not become diseased.
I thank you in advance for any and all efforts to formally validate my experiences and research and to make this common medical knowledge known everywhere by ALL medical professionals and finally to their patients.